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Figures 2/3 Gordon et al (2016) A fine balance of synaptophysin levels underlies efficient retrieval of synaptobrevin II to synaptic vesicles (PLOS ONE)
Data for Gordon et al (2016) PLoS One - "A fine balance of synaptophysin levels underlies efficient retrieval of synaptobrevin II to synaptic vesicles".
* Figures 2 and 3 – TIFF stacks displaying the synaptobrevin ...
Figure 4 Gordon et al (2016) A fine balance of synaptophysin levels underlies efficient retrieval of synaptobrevin II to synaptic vesicles (PLOS ONE)
Data for Gordon et al (2016) PLoS One - "A fine balance of synaptophysin levels underlies efficient retrieval of synaptobrevin II to synaptic vesicles".
* Figure 4 - TIFF stacks displaying the synaptobrevin II-pHluorin ...
NMJ-morph
NMJ-morph is an ImageJ-based package for morphometric analysis of the neuromuscular junction (NMJ).
Post-mortem brain analyses of the Lothian Birth Cohort 1936: Extending lifetime cognitive and brain phenotyping to the level of the synapse: Data set from publication Henstridge et al 2015 Acta Neuropath Comms
Non-pathological, age-related cognitive decline varies markedly between individuals and places significant financial and emotional strain on people, their families and society as a whole. Understanding the differential ...
Figure 1 Gordon et al (2016) A fine balance of synaptophysin levels underlies efficient retrieval of synaptobrevin II to synaptic vesicles (PLOS ONE)
Data for Gordon et al (2016) PLoS One - "A fine balance of synaptophysin levels underlies efficient retrieval of synaptobrevin II to synaptic vesicles".
* Figure 1 – TIFFs displaying fluorescent images after immunostaining ...
SUPERSEDED: NMJ-morph
Please see the updated NMJ-morph at https://doi.org/10.7488/ds/1490 . NMJ-morph is an ImageJ-based package for morphometric analysis of the neuromuscular junction (NMJ).
Non-fibrillar oligomeric amyloid-β within synapses: Data set from publication Pickett et al 2016 J Alz Dis
Alzheimer’s disease (AD) is characterized by memory loss, insidious cognitive decline, profound neurodegeneration and the extracellular accumulation of amyloid-beta (Ab) peptide in senile plaques and intracellular accumulation ...
Human tau increases amyloid beta plaque size but not amyloid beta-mediated synapse loss in a novel mouse model of Alzheimer’s disease: Data set from Jackson et al 2016 EJN
Alzheimer’s disease is characterized by the presence of aggregates of amyloid beta (Aβ) in senile plaques and tau in neurofibrillary tangles, as well as marked neuron and synapse loss. Of these pathological changes, synapse ...
